Klebsiella pneumoniae

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      The prostatitis syndrome is not a figment of your imagination as insurance companies and many ignorant practitioners would like you to believe. For every effect there is an exact cause!  Despite what people tell you, there is a damn good reason why 40% of all prostatitis cases are not resolved with antibiotics alone. When this cause remains hidden, the wrong treatment needlessly perpetuates suffering for decades if not the rest of people’s lives. If you are reading this address you already realize chronic infection will not just go away on its own or through the use of some antibiotic cure all. When patients don’t respond to antibiotics there is always a reason why. The prostatitis syndrome is caused by various bacteria, funguses, and viruses, as well as, mixed infections. Antibiotics can’t suppress the plunder of a viral or fungal infection, likewise, they are virtually useless on severely resistant bacteria.

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 A Brief Summary of Androgen Scientific Research Group activity:

      The Androgen Scientific Research Groupwas created in 1990 with the purpose of rendering assistance to men suffering chronic inflammatory processes in their reproductive sexual glands: prostate and/ or seminal vesicles. Dr. Vasily Bairaktar, Doctor of Medical Science at Odessa University, leads the scientific and clinical direction of the group. Dr. Bairaktar with his colleagues concentrate on detailed diagnosis of prostatitis, as well as, the use of new, modern, highly effective technologies in treatment for patients suffering chronic inflammation in the prostate gland and seminal vesicles.

     The Group «Androgen» consists of the top specialists of the major medical schools of the city of Odessa. The following specialties are represented in the Group: Immunologists, microbiologists, virologists, mycologists, experts in ultrasonic diagnostics, experts in clinical and biochemical laboratory diagnostics, urologists, endocrinologists. The powerful combination of such medical specialties helps the group achieve success where doctors with single specialties are have failed.

The tasks of the Scientific Research Group- «Androgen»Activities of the group

  1.     Diagnose the latent inflammatory processes in the prostate and seminal vesicles.

  2.     Detailed diagnostics: isolation of the specific infective agent in prostatitis or seminal vesiculitis (bacterium, viruses or fungi)

  3.     Define the type and form of the infective agent in prostatitis or seminal vesiculitis.

  4.     Define the condition of the cellular and humoral immunity link of patients.

  5.     Structural — examine the morphological features of the prostate using ultrasonic diagnostics.

  6.     Preparation of immunogen conjugates from viral, bacterial and fungal agents in the prostate or seminal vesicles. Each conjugate is custom-made for each patient.

  7.     Treatment with immunogen conjugates while thoroughly monitoring the immunological reactions of the patient.

Methods of assessment and diagnosis of inflammation in the prostate and seminal vesicles.

  1.     Bacteriological and mycological diagnosis with the use of selective nutrient media intended for the selection of bacteria in semen from subjects suffering from prostatitis and seminal vesiculitis.

  2.     Diagnosis of Trichomonosis using selective nutrient media in cultures.

  3.     Virological diagnosis using nutrient media intended for selection of the viruses from semen of subjects suffering prostatitis and seminal vesiculitis.

    1. Isolation of Herpes Simplex Virus, Papilloma viruses, Adeno viruses, Entero viruses and other viruses and Chlamydias, from secretions of men’s reproductive sexual glands (semen) grown on special cell line cultures. (Note: Serological virology methods fail to isolate the live viruses 75% of the time, and therefore it is essential to uses special cell lines.)

    2. Immunofluorescent analysis with specific Chlamydias, Herpes Simplex Virus, Mycoplasma, Ureaplasma and other virose-bacterial antibodies for the presence of Chlamydias, Herpes Simplex Virus and other virose-bacterial antigens in semen of men.

    3. PCR (serological reaction) diagnostic.

    4. Immunofermentation analysis of blood for presence of antibodies and definition of their titer to:

      Titer of antibody in blood to some viral, bacterial, fungal infection:

      1. Cytomegalovirus;

      2. Herpes virus type-6;

      3. Herpes simplex virus type 1 and 2

      4. Epstein-Barr virus;

      5. Adeno viruses;

      6. Papilloma virus;

      7. Papova viruses;

      8. Chlamydia trachomatis; pneumoniae;

      9. Mycoplasma hominis;

      10. Ureaplasma urealiticum

      11. Toxoplasma gondii

      12. Trichomonas vaginalis

    5. When it is necessary we perform a subcutaneous test with chlamydial antigens to detect the presence of a chlamydial infection.

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Traditional Methods of treatment of Prostatitis and Seminal vesiculitis are with weak therapeutic effect.

      Traditional methods of treatment of inflammatory processes in the prostate and seminal vesicles by antibiotics are still widely used. Treatment by antibiotics is only justified in acute cases, where it is a matter of life or death. The repeated use of antibiotics for a chronic inflammatory process in the prostate and seminal vesicles destroys biofilm bacteria such as Bifidumbacterium, Escherichia coli, Lactobacillus in the intestines, and so, frequently causes disbacteriosis in the intestines.

      In disbacteriosis the balance of bacterial types in the patient is broken, and so, biofilm bacteria perish. In their place pathogenic bacteria and viruses take hold. Over the years parasites populate the intestines and other parts of the body rendering a negative influence on many of the patient’s normal systems. This cycle can go on for decades without amelioration.

      This has become our working concept. It is confirmed by our research and the research of our colleagues. Typically chronic prostatitis and seminal vesiculitis is treated by using several courses of antibiotics like: Cipro, Ciprolet, Ciprobid, Ciprobay (Cyprofloxacin), Cefobid, Ofloxacin (Tarivid), Trovan, Norfloxacin, and other types of antibiotics like Metronidazole (Flagyl, Tinidazol) are also widely used. The Bacteria have the ability to form a resistance — «to get used» to the repeated actions of antibiotics, therefore frequent use reduces the therapeutic effect to zero. Our research has shown that the use of antibiotics drastically hinders the function of the liver, kidneys, blood and most importantly suppresses the protective functions of the immune system.

     We have found it possible to avoid the application of antibiotics all together for our treatment of chronic prostatitis and chronic vesiculitis. The use of antibiotics for chronic inflammatory processes does not eradicate pathogenic bacteria in a patient. Antibiotics only delay the duplication and growth of bacteria, and so, do not provide a permanent solution. Once antibiotics are stopped the same bacteria again multiply in the patient’s body and cause a relapse of prostatitis and vesiculitis. For this reason, antibiotics are prescribed over and over again over many years and still do not solve the problem. It is important to note that antibiotics do not influence viruses at all. You can take antibiotics for the rest of your life, but it will do you no good if your symptoms are caused by a virus.

      During treatment of chronic prostatitis and vesiculitis we use physiotherapeutic procedures as an auxiliary factor to increase the effectiveness of our treatments.

      We have included a combination of physiothera peutic procedures and use these if necessary:

1) Rectal ultrasonic medical procedures on the areas of prostate and seminal vesicles is useful in reducing calcification in prostate and normalizing the overall structure.

2) Weak medical infrared laser over the lower abdomen. It helps stimulate the immunoprotective functions and suppress pathogens.

3) Medical amplitude impulses (stimulating therapy). Suppress inflammation, reduce pain and normalize nervous system function in pelvic area.

4) Medical electrophoresis (iongalvanization). This procedure allows the diffusion of certain substances in the body (e.g. ZnSO4 (restores nervous system function in pelvic area), KI (anti-inflammatory), Novocain, etc.) through the lower abdomen and lower back with the aid of a static electric field.

       The use of such physiotherapeutic procedures is necessary especially when there are inflammatory stones in the prostate, and also:

1) Helps the infiltration of the prostatic capsule (electrophoresis),

2) When the prostatic tissue is not homogeneous,

3) When there is calcification in the prostate. (Note: After many years of a chronic infection without proper treatment the tissue of the prostate changes. The tissue no longer properly secretes necessary fluid.)

      The use of the physiotherapeutic procedures helps us to restore the morphological structures of the prostate. As a result, the reduced secretory function is restored to the prostate and seminal vesicles.

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New concepts and technology for treatment of prostatitis and seminal vesiculitis in young men:

      Our new technological treatment of prostatitis and seminal vesiculitis is based on the fundamental understanding of clinical immunology. Immunogen conjugates, made by the Androgen Scientific Research Group- are used to influence the action on specific pathogens involved in prostatitis and seminal vesiculitis this stimulates the specific immune response of the patient. In the process, pathogenic agents of prostatitis and seminal vesiculitis are eradicated.

       The use of Immunogen conjugates facilitate the stabilization of normal microflora in the inflamed area. This provides the patient a long period of resistance to microorganisms for which we have provided immune protection. Our use of immunogen conjugates against agents in prostatitis and vesiculitis represents a new and highly effective method of treatment that is far superior to antibiotics. We carry out detailed virological, bacteriological and mycological (fungal) diagnostics on semen to identify the types of bacteria, fungi or viruses responsible for the infection. There is no guess work involved in our diagnostic methods, all treatments are based on the isolation of specific pathogens. We extract, select, and isolate the infective agent from semen, urine, and EPS (Express Prostatic Secretions). After we have selected pathogenic bacteria, fungi or viruses alive we prepare high immunogen conjugates from these isolated microorganisms for each patient.

      The technological process of preparation of immunogen conjugates depends upon the type of infection for the specific patient. This process takes time approximately as follows:

  1. For bacteria – preparation continue — 1 month;

  2. For fungi – 1-2 month (It depends on the kind of fungus);

  3. For viruses – 2-3 months (It depends on the kind of viruses).

       Before beginning treatment we do a detailed assessment of all aspects of the patient’s immune system. We carefully estimate the condition of the immune system by checking the cell and humoral immunity that indicates natural resistance. Taking into account specific immunological problems, we choose the appropriate type of immunological correction. If a patient has autoimmune diseases or if the patient is sensitive to the tested bacterial, viral and fungal antigens, we carry out desensitization with specific antigens.

      Only after desensitization do we begin generating active specific immunity for the microorganisms involved in the patient’s disease.

      We begin manufacture of the immunogen conjugates only after the patient pays the cost of Chemical Reagents and Nutrient Media for bacterial, fungal or viruses cultures. The reagents and medias are an indispensable part of generating high immunogen conjugates. The course of treatment consists of 3 intramuscular injections with an interval of 10 days between injections.

       Contra-indications for use of our immunogen conjugates are allergies or predisposition to allergic diseases.

       Our treatment is individualized and entails significant labor cost in the preparation of immunogen conjugates.

       As a result, we can only treat a small number of patients. Typically, a patient stays for three weeks, and in this period he is the only patient treated. This allows us to monitor each patient’s progress on a continuous basis so we can direct all our efforts on curing him. The nature and method of our treatment involves significant cost, and therefore, is addressed only to persons with the necessary financial means. The cost of manufacture of high immunogen conjugates directly depends on the types of microorganisms isolated from the individual patient during detailed diagnostics. It is calculated in each specific case individually.

      Our methods can resolve the problems of individuals who have been unsuccessfully treated for prostatitis and seminal vesiculitis, because our treatments overcome the problem of antibiotic resistance and serve to effectively eradicate viral causes that thus far have not been treated.

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Our conceptual approach to the treatment of recurring prostatitis due to viral, bacterial or fungal infections.

    Our new concept has been carefully refined and tested for over ten years.:

    Our new conceptual approach focuses on the treatment of chronic prostatic infection by viral, bacterial or fungal pathogens without the use of antibiotics.

    Our purpose: Forming specific immunity protection to the chronic relapse of prostatitis caused by viral, bacterial or fungal infections based on individual immunoreactivity of CP sufferers.

To achieve our purpose we perform the following tasks:

  1.     Check the status of cellular and humoral immunity in general. Identify any allergic sensitization to specific viruses-bacteria-fungi to antigens. Determine status of cells that determine natural resistance of the body (by measuring cell toxicity from activity of » killer cells (CD+25) and functional status of macrophages (mononuclear cells and phagocytes)).

  2.     From the analysis of the immune status of the patient we can determine the required immunocorrective therapy. We use medicines which primarily influence the T cell (CD3+) immunity link, humoral immunity, and natural resistance.

  1. If the patient is allergic (highly sensitive) to viral, bacterial or fungal infective agents we follow a specific desensitization therapy as a first step, prior to the onset of conjugate therapy.

  2.     After immune tests we can determine if the systemic patient immunity is satisfactory, we proceed with specific immune therapy employing specific viral, bacterial or fungal immunogen conjugates once we deem the function of the immune system to be normal. These conjugates generate significant amounts of antibodies, specific to the isolated pathogen.

  3.     Taking into account the initial level of immune status of the patient we make individual selection of dosage of immunogen conjugate (by checking the reaction of CD-3 (T-cells) and other effectors for viral, bacterial or fungal immunogen conjugates).

  4.     In some cases, when we can not receive the necessary level of desensitization and immune correction (by a series of antigen shots) we use a combination of low doses of immunogen conjugates, and immune modulators (alpha — 2 recombinant interferon etc.). The result is an increase in the receptor cells that help to reduce inflammation, while generating sufficient number of antibodies to fight the pathogens.

  5.     We perform an immune reaction control test to monitor the status of the patient’s body and determine the rate of the subsequent introduction of viral, bacterial or fungal immunogen conjugates.

  6.     We administer physio-therapeutical procedures before and after specific immune therapy when it necessary.

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Terms for detailed diagnostics and treatment of inflammation of male sexual glands.

1.  Detailed diagnostics — term – 1 to 3 days, depends on type of tests.

2. Preparation of immunogen conjugate from bacteria — term of preparation — 1 month from the date of payment and purchse all necessary chemical reagents and medias.

3. Preparation immunogen conjugate from fungus- term of preparation — 2 month from the date of payment and purchse all necessary chemical reagents and medias.

 4. Preparation immunogen conjugate from Chlamydia trachomatis and Chlamydia pneumoniae — term of preparation — 2 month from the date of payment and purchase all necessary reagents and medias.

5. Preparation immunogen conjugate from Mycoplasma — term of preparation — 2 month from the date of payment and purchase all necessary reagents and medias.

6. Preparation of immunogen conjugate from Ureaplasma urealiticum — term of preparation 2 month from the date of reception all necessary reagents and payment.

7. Preparation immunogen conjugate from viruses — term of preparation 2-3 month from the date of reception all necessary reagents and payment.

8. Preparation of immunogen conjugate from Herpes Simplex virus type 1- or type 2- :

9. From Herpes virus virione- term of preparation 1-1,5 month from the date of reception of all necessary reagents and payment. Time for preparation and cost depends on viruses strains. This conjugate is prepared from virione (the body of the virus).

10. Subunit conjugate (virus free) of Herpes virus is an improved variant without including virione. Term of preparation 3 month from the date of payment and purchase necessary for preparation reagents and medias. This conjugate is prepared from cell culture which infected with alive Herpes Virus and then extracted from these infected cell culture. Glycoproteids from surface of Herpes viruses extracted. This process include affinity chromatography. Conjugate is highly immunogen for specific treatment of Herpes virus sufferers. Using this technology, the conjugate does not have present viruses but only the viral glycoproteids and antigens, therefore is a safer.

11. Gene ingeneering Herpes virus conjugate which includes only immunogen part of Herpes virus.

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  Published scientific article: » Specific immune protection to Klebsiella pneumoniae and Enterococcus faecalis in the chronic prostatitis sufferers» read at URL address:

Diagnosis and treatment for urogenital Сhlamydiosis

       Chlamydia trachomatis and Chlamydia pneumoniae characterized by intracellular localization and 15 sero species and variants. For sexual organs, infections with sero species D, E, F, G, are important. Chlamydia trachomatis and Chlamydia pneumoniae are hard to isolate because they live intercellularly in the body. Special cell line virological cultures are needed to grow these Chlamydias outside of the body. The body can not eradicate Chlamydia trachomatis and Chlamydia pneumoniae infections on its own. It is not capable of secretion of protective antibodies, and therefore there is no hope for spontaneous cure from this chronic infection. The immune system can only secrete seroconversive antibodies, which do not give any real help or protection against the caustive Chlamydial agent.

      Doctors often try ameliorate this problem with antibiotics and non-specific stimulation of the immune system. This treatment can not provide lasting relief to patients because Chlamydia trachomatis and Chlamydia pneumoniae often become completely resistance to antibiotics. Therefore after one or two courses of antibiotics resistance develops and further use of antibiotics is useless. Antibiotics only produces temporary results. While the patient is taking antibiotics, improvement is observed, but after antibiotics are stopped Chlamydia continues on its course unscathed, the only difference being that now the Chlamydia is resistent to antibiotics. Furthermore, antibiotics suppress the cellular link of the immune system. Frequently mixed Chlamydia and viral infections are present. In these cases antibiotics do not even have the capability to help, they only make the problem worse. Chlamydia is not a typical bacterial infection, and so does not respond to antibiotics like a normal bacterias do. Chlamydias have many qualities of a viruses, and so, respond better to treatment normally used to treat viruses.

      The use of antibiotics leads to disfunction of bacterial balance in intestines. Antibiotics disturb and may even eradicate this intestinal flora — a phenomenon known as disbacteriosis. This intestinal flora plays an essential role in the body metabolism and thus affects the immune system balance. Eradicated intestinal bacteria are replaced by pathogenic viruses, fungus, and bacteria causing a mixed infection in the body. Our group avoids the use of antibiotics all together. We extract chlamydial antigens (thermoresistent protein) from the particular infective agent. We provide specific immunoprotection to Chlamydia trachomatis or Chlamydia pneumoniae by creating conjugates from the derived antigens.

                       Diagnosis of Chlamydiosis :

  1. solation of a live Сhlamydias from men in the secretions of sexual glands (semen, urine, and EPS) using special cell line cultures.

  2. Immunofluorescent analysis specific to Сhlamydia trachomatis and Сhlamydia pneumonia antibodies for the presence of chlamydial antigen in semen, urine, and EPS of men (only as auxiliary and approximate method).

  3. Immunophermentative tests of blood for presence of antibodies and definition of their titer to Сhlamydia trachomatis and Сhlamydia pneumoniae.

  4. PCR (To identify DNA) test for Chlamydia trachomatis and Chlamydia pneumoniae.

  5. Sometimes if necessary, make a intracutaneous test introducing the chlamydial antigene to determine sensitivity (allergic response) of the patient to Chlamydia trachomatis or Chlamydia pneumoniae.

For treatment we prepare two variants of Chlamydial conjugates:

1) Chlamydia conjugates of short action, which we use for gradual desensitizing therapy to Сhlamydia trachomatis or Сhlamydia pneumoniae.

2) Chlamydial conjugate of long actions, which we use for the patients suffering from chronic chlamydiosis. This Chlamydial conjugate differs by concluding it in sorbent, which creates good base for chlamydial thermoresistent protein and gives strong adjuvantive effect.

     Before we begin desensitizing therapy to Сhlamydia trachomatis or Сhlamydia pneumoniae on the patient, we do all immune tests and analysis is carried out in strict order. The immunologic parameters we study in detail include the definition of cellular condition and humoral links of the immune system.

     If the patient has low phagocyte activity of neutrophils, we raise the level of this parameter in blood through appropriate therapy before administering conjugates. People with very low phagocyte activity we can not accept into treatment, until they first normalize this condition.

    We take into account the following parameters of blood to determine if patients are suitable for our treatment:

  1. WBC (white cells concentration of blood;

  2. ESR60 (Speed of sedimentation red cells concentration of blood;

  3. Lymphocytes;

  4. CD+3 (T- lymphocytes);

  5. CD+4 (T- chelpers);

  6. CD+8 (T- supressors);

  7. Ratio CD4/CD8;

  8. CD+19 (B- lymphocytes);

  9. CD+25 (T- killers);

  10. Phagocytical activity neutrophils (Phagocitosis);

  11. Response T- lymphocytes to chlamydia trachomatis antigene.

  12. Immunoglobuline – A;

  13. Immuneglobuline – G;

  14. Immuneglobuline – M;

  15. Immuneglobuline – E.

  16. The titer of antibodies in blood to Chlamydia trachomatis and Chlamydia pneumoniae for IgA; IgG; IgM (need in concrete antibody titers).

    For example: IgA – 16; IgG – 32; IgM – 16.

      If the cellular link of a patient`s immune system is disbalanced, we carry out immunocorrection using non-specific immunomodulators. Only after a desensitizing process can we create a specific immunomodulation.

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Diagnostics and treatment for Herpes Simplex virus type 1 and type 2 in men

     Herpes simplex virus can only be isolated in special cell line cultures. Diagnosis for Herpes Simplex virus is carried out by our research group through the following criteria:

  1.   Isolation of the Herpes Simplex virus from infected men sexual gland secretions (semen, EPS, urine) using special cell line cultures.

  2.  Immunofluorescent analysis which is used to check for the presence of Herpes Simplex virus antigen in semen, urine, and EPS.

  3.  Immunophermentative tests of blood to check the presence of antibodies and to define their titer for Herpes Simplex virus type 1 and type 2.

  4.  PCR test for Herpes Simplex virus type 1 and type 2.

  5.  Sometimes if necessary, we monitor the response following an intracutaneous introduction of Herpes Simplex virus type 1 and type 2 antigens to determine the sensitivity (allergic response) of the patients to Herpes Simplex virus type 1 and type 2 antigens.

            For treatment we prepare two variations of Herpes Simplex virus conjugates:

  1.   Herpes Simplex virus type 1 & 2 or separately for type 1 and separately for type 2 of short action, which we use as a gradual desensitizing therapy

  2.   Longterm action conjugates are also produced to provide more lasting protection.

     Herpes Simplex virus sub unit high immunogen conjugate – improved variant, without containing any viral virions, term of preparation 4-5 months. This Herpes virus conjugate differs by concluding it in sorbent, which creates good base for Herpes virus glycoprotein and gives good and strong adjuvantive effect.

      Before we begin conjugate treatment , we perform necessary immune tests. We monitor the immune system to define the cellular condition and humoral links of the immune system. If the patient shows low results for phagocytical activity of neutrophils, we recommend treatments to raise these parameters. If low parameters persist following corrective treatment, we can not proceed with high immunogen conjugate therapy.

           We consider the following parameters of blood:

              —  WBC (white cells concentration of blood;

             —  ESR60 (Speed of sedimentation red cells concentration of blood;

            —  Lymphocytes;

            —  CD+3 (T- lymphocytes);

            —  CD+4 (T- chelpers);

            —  CD+8 (T- supressors);

            —  Ratio CD4/CD8;

            —  CD+19 (B- lymphocytes);

            —  CD+25 (T- killers);

            —  Phagocytiar activity of neutrophils (Phagocytosis);

           —  Response T- lymphocytes to chlamydia trachomatis antigen.

          —   Immuneglobuline — A;

         —   Immuneglobuline — G;

         —   Immuneglobuline — M;

         —   Immuneglobuline — E.

     The IgG titer of antibodies in blood to Herpes simplex virus type 1 and type 2;

     If the cellular link of a patient`s immune system is disbalanced, we carry out immunocorrection using non-specific immunomodulators. Only after a desensitizing process can we create a specific immunomodulation.

     Synthetic antiviral remedies help the body produce interferone. However, patients with chronic infection have a broken immune link which makes it impossible for the body to produce its own interferone. For this reason synthetic antiviral medicines can not help. Natural recombinant interferone provides direct antiviral action.

   The medical effect from anti-virus pharmaceutical remedies (Acyclovir (Zovirax); Valacyclovir (Valtrex); Famciclovir (Famvir), and others is not significant.

   Another undesirable result of the use of antiviral remedies is the depression of cellular link of the immune system and other very bad result to the body of the patient.

   We have enough clinical material with good medical results for the use of high immunogen conjugates from Herpes simplex virus type 1 and type 2 (separately or combined), without any negative results and actions.

   We are ready to share it with patients from other countries who are suffering to Herpes virus infection.

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  List of questions whose answers we require prior to initiating E-mail contact:

                             Please answer the following:

  1. Your exact legal name?

  2. Your real age?

  3. What country and city you reside in?

  4. Are you single or married?

  5. What diagnostics and treatments did you go through and when?

  6. What kind of sexual activity to you engage?

  7. What prostatitis symptoms do you currently have?

  8. In present time do you have sexual partner or not?

  9. Do you have protection with condoms or not?

  10. Have you been tested for HIV – If so, what is the result?

  11. Had/have any STDs? If you had/have, what type of STDs specifically?

  12. In present time do you have any pain in prostate area after ejaculation or not?

  13. In details: what kind of infections do you have in semen and urine bacterial species and for how long it continue.

  14. Have/had you any color change of your sperm? Any sign of blood in the sperm or not at all?

  15. Have/had you irritation in penis before and after ejaculating?

  16. Have/had you any itching, little red dots, pimples or spots on the head of penis?

  17. Have/had you any allergic reactions to food? If so, please specify which products you are allergic to?

  18. Have/had you any allergic reactions to remedy? If you have allergic reaction specify which remedy.

  19. Have/had any ultrasound tests of your prostate? If you have test results, send them by E-mail.

  20. Have you had Iimmune test of your blood? If you have these results, send them to us by E-mail.

  21. Had you had any tests of your sperm? If you have these results, send them to us by E-mail.

  22. Had you any bacteriological tests of your sperm? If you have these results, what species of bacteria isolated from your sperm sample.

  23. Do you have proper funds available to come to Odessa for detailed diagnostics and treatment?

      This is tough subject to discuss over the Internet, but you can be sure that all correspondence abetween us will be strictly confidential.